The Role of STAT 3 in Tissue Fibrosis
Keywords: Astrocytes; C-terminal transactivation; DNA binding; Idiopathic pulmonary fibrosis (IPF); IgA; Immunofluorescence; Keloid; Receptors; Renal Interstitial Fibrosis; STAT3; Spinal Cord Injury; aging; allergic responses; autoimmune; bleomycin injury; bone marrow; carbon tetrachlorideinduced; central nervous system; chronic autoimmune diseases or rachitis; cirrhosis; coiled-coil; collagen deposition; cytokines; cytokines/growth factors; cytoplasmic transcription; diabetic; epithelial cells; extracellular matrix; fibroblasts; fibrogenic actions; fibroproliferative diseases; glial cells; glomerular sclerosis; glomerulonephritis; growth factor, (TGF); hardening; hematopoetic factors; hemodynamic; hepatic stellate cells; hyperactivation; hyperfiltration; hypertension; inflammatory cells; interstitial edema; kidney; leukocytes; metabolic disorders; migration; motor recovery; myeloid metaplasia; myocardial infarction; nephron; neutrophils; obstructive nephropathy; organ failure; oxidative stress; parasitic diseases; pathogenesis of tissue; phosphorylation; platelet derived growth factor (PDGF); proliferation; protein-protein interactions; skin lesions; smooth actin; syndrome; tissue fibrosis; traumatic injuries; tyrosine kinase; unilateral ureteral obstruction (UUO); urinary albumin excretion; visceral epithelium
Document Type: Research Article
Publication date: January 1, 2011
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