Cellular and molecular mechanisms in transplant arteriosclerosis (Review).
Although significant progress has been made towards the understanding of cellular and molecular mechanisms underlying the pathogenetic pathways of transplant arteriosclerosis, its knowledge is still not comprehensive. Nevertheless, experimental and clinical studies have enabled us to discover some of the complex processes involved in the progression and evolution of transplant arteriosclerosis. Despite the advances in transplantation immunology and atherosclerosis research, transplant arteriosclerosis still remains a major cause of allograft failure. A curative treatment, in order to inhibit or at least modify the development of transplant arteriosclerosis, is urgently needed. This review article highlights some of the more recent aspects of cellular and molecular pathology of transplant arteriosclerosis that may add to our current and future diagnostic and curative interventions.
No Reference information available - sign in for access.
No Citation information available - sign in for access.
No Supplementary Data.
No Article Media
Document Type: Research Article
Affiliations: Department of Pathology, University Hospital, S-751 85 Uppsala, Sweden.
Publication date: January 1, 1998
More about this publication?
- The International Journal of Molecular Medicine is a monthly, peer-reviewed journal devoted to the publication of high quality studies related to the molecular mechanisms of human disease. The journal welcomes research on all aspects of molecular and clinical research, ranging from biochemistry to immunology, pathology, genetics, human genomics, microbiology, molecular pathogenesis, molecular cardiology, molecular surgery and molecular psychology.
The International Journal of Molecular Medicine aims to provide an insight for researchers within the community in regard to developing molecular tools and identifying molecular targets for the diagnosis and treatment of a diverse number of human diseases.
- Editorial Board
- Information for Authors
- Submit a Paper
- Subscribe to this Title
- Information for Advertisers
- Terms & Conditions
- Ingenta Connect is not responsible for the content or availability of external websites