Skip to main content

Neonatal skin barrier: structure, function, and disorders

Buy Article:

$43.00 plus tax (Refund Policy)


The development of the human skin from intrauterine to extrauterine life is a balletic interplay of maturing layers and interlocking structures. We discuss this transition and then branch out to touch on issues of premature infant as well as neonatal skin care. Disruption of the barrier function due to toxins and development errors are expounded upon. Staph scalded skin syndrome, collodion membrane, bullous congenital ichthyosiform erythroderma, autosomal recessive ichthyosis (lamellar and congenital ichthyosiform erythroderma), and harlequin fetus are used as examples of these disruptions. Discussion of therapy with the authors’ experience highlights each disease.
No References
No Citations
No Supplementary Data
No Article Media
No Metrics

Keywords: Neonatal skin; Staphylococcal scalded-skin syndrome; barrier function; bullous and non-bullous congenital ichthyosiform erythroderma; collodion membrane; harlequin fetus; lamellar ichthyosis; nosocomial infections; premature infant skin

Document Type: Research Article

Affiliations: Pediatrics, Sparrow Hospital, Lansing, MI

Publication date: 2005-03-01

  • Access Key
  • Free content
  • Partial Free content
  • New content
  • Open access content
  • Partial Open access content
  • Subscribed content
  • Partial Subscribed content
  • Free trial content
Cookie Policy
Cookie Policy
Ingenta Connect website makes use of cookies so as to keep track of data that you have filled in. I am Happy with this Find out more