Authors: Bozec, A.; Lassalle, S.; Hofman, V.; Ilie, M.; Santini, J.; Hofman, P.
Source: Current Medicinal Chemistry, Volume 17, Number 30, October 2010, pp. 3449-3461(13)
Publisher: Bentham Science Publishers
Keywords: Autoimmune Thyroid Diseases; B-lymphocytes; BRAF mutation; CD8+; Chronic infection; Classical TAM activation (TAM1); Dysregulation; Hashimoto's thyroiditis; Hashimoto's thyroiditis (HT); Inflammation; Inflammation-Induced Carcinoma; Macrophages; Medullary thyroid carcinomas; RET/PTC rearrangement-activation; Somatic mutations; T-CD4+; Therapeutic Potential; Thyroid Gland; Thyroid carcinoma; angiogenesis; angiogenesis and metastasis; anti-inflammatory; apoptosis; autophagy; cancer; carcinogenesis; chemokines; chronic inflammation; cytokines; dendritic cells; gastrointestinal level; genetic alterations; hypothyroidism; immune cell microenvironment; interleukin IL-10; leukocyte adhesion; macrophages (TAM); mast cells; membrane-bound small G-protein RAS; metastasis; mitogenic, angiogenic or motogenic factor; mucosa-associated lymphoid tissue (MALT); multicentric tumors; multinodular goiter; natural killer; neoplastic; neoplastic transformation; neutrophils; pancreatic cancer; papillary thyroid carcinoma (PTC); proinflammatory cytokines; prostaglandin E2; thyroid; tumor cell proliferation; tyrosine kinase membrane; vascular endothelial growth factor (VEGF)
Document Type: Research Article
Affiliations: Laboratory of Clinical and Experimental Pathology, Pasteur Hospital, BP69, 06002 Nice cedex 01, France.
Publication date: October 1, 2010